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Platelet Aggregation       Article     History   Tree Map
  Encyclopedia of Keywords > Policosanol > Platelet Aggregation   Michael Charnine

Keywords and Sections
PLATELET PLUG
PLATELETS RESULT
PLATELET FACTORS
INHIBITORY EFFECT
VASODILATION
SMOKING-INDUCED PLATELET AGGREGATION
PLASMA INHIBITOR
ADP-INDUCED PLATELET AGGREGATION
CLUMPING
DEFECTIVE PLATELET AGGREGATION
VASOCONSTRICTION
PLATELET AGGREGATION INHIBITORS
HUMAN PLATELET AGGREGATION
ROLE
LEADING
VITAMIN
DIAGNOSIS
ADMINISTRATION
STUDIES
GARLIC
RESULTING
PROCESSES
THROMBOSIS
GROUPS
RESULT
BLOOD VESSEL
BINDING
TYPE
HYPERCHOLESTEROLEMIA
METABOLITES
FIBRIN
ARGATROBAN
CARDIOVASCULAR DISEASE
PRAVASTATIN
THROMBUS FORMATION
THROMBIN
CLOT FORMATION
ACTIVATION
BLOOD COAGULATION
TREATMENT
PROSTACYCLIN
ETORICOXIB
LIPID PROFILE
ARACHIDONIC
EFFECTS
HUMANS
Review of Short Phrases and Links

    This Review contains major "Platelet Aggregation"- related terms, short phrases and links grouped together in the form of Encyclopedia article.

Definitions

  1. Platelet aggregation is a predominantly physical process, whereby platelets clump together, and coagulation is a cascade of biochemical enzyme reactions.
  2. Platelet aggregation was greater in hypercholesterolemia than in normocholesterolemia before treatment and was altered by fluvastatin.
  3. Platelet aggregation was determined at baseline and after 15 days of treatment.
  4. Platelet aggregation is increased in patients with MS, while fibrinogen levels remain similar to controls.
  5. B, Platelet aggregation was significantly reduced after treatment with fluvastatin but not with colestimide.

Platelet Plug

  1. Platelet activation is accompanied by platelet shape change, release of cytoplasmic granule constituents, and platelet aggregation to form a platelet plug.

Platelets Result

  1. The glycoprotein IIb-IIIa receptors also bind fibrinogen, and fibrinogen bridges between platelets result in platelet aggregation.

Platelet Factors

  1. It causes a time and dose-dependent inhibition of platelet aggregation and release of platelet factors, as well as a prolongation of bleeding time.

Inhibitory Effect

  1. An inhibitory effect of policosanol on platelet aggregation and cerebral ischemia in animal models has been reported.

Vasodilation

  1. Dipyridamole is a drug that inhibits platelet aggregation and causes vasodilation.

Smoking-Induced Platelet Aggregation

  1. Inhibition of smoking-induced platelet aggregation by aspirin and pycnogenol.

Plasma Inhibitor

  1. A plasma inhibitor of platelet aggregation in patients with Argentine hemorrhagic fever.

Adp-Induced Platelet Aggregation

  1. The results were expressed as the percent changes in nitrotyrosine-specific staining of platelets by ADP-induced platelet aggregation in each group.

Clumping

  1. Platelet aggregation: The clumping together of platelets in the blood.

Defective Platelet Aggregation

  1. Patients on aspirin have an increased bleeding time and defective platelet aggregation.

Vasoconstriction

  1. Guava leaf extract did not affect bleeding times, it stimulated vasoconstriction and platelet aggregation but it inhibited blood coagulation.

Platelet Aggregation Inhibitors

  1. Anti-platelet drugs: Anti-platelet drugs, also called platelet aggregation inhibitors, have been used to prevent platelets from forming clots.

Human Platelet Aggregation

  1. Dehydroepiandrosterone inhibits human platelet aggregation in vitro and in vivo.

Role

  1. Another target of pharmacologic intervention in coagulation involving platelets is the role of GPIIb-GPIIIa in fibrinogen-induced platelet aggregation.

Leading

  1. The shear stress and the agonists in the membrane activate platelets, leading to platelet aggregation.

Vitamin

  1. Normalization of hyperhomocysteinemia with vitamin B(12) and reduction of platelet aggregation with n-3 fatty acids.

Diagnosis

  1. Chong BH, Burgess J, Ismail F. The clinical usefulness of the platelet aggregation test for the diagnosis of heparin-induced thrombocytopenia.

Administration

  1. Inhibition of platelet aggregation is detected within 2 days of administration with 250 mg b.i.d.

Studies

  1. Isenhart CE, Brandt JT. Platelet aggregation studies for the diagnosis of heparin-induced thrombocytopenia.

Garlic

  1. Platelet aggregation inhibitors include aspirin, green tea, ginkgo, garlic, and vitamin E (Bossavy et al.

Resulting

  1. Diabetes also impairs platelets function therefore resulting in platelet aggregation.

Processes

  1. This response involves two intertwined processes: platelet aggregation and coagulation.

Thrombosis

  1. Platelet aggregation and therefore thrombosis is accelerated in patients with elevated triglyceride levels.
  2. Defective platelet aggregation and increased resistance to thrombosis in purinergic P2Y(1) receptor-null mice.

Groups

  1. There was no significant difference in fibrinogen and fibrinopeptide A levels, nor in 'in vitro' tests of platelet aggregation between the groups.

Result

  1. The change in resistance between the electrodes which takes place during platelet aggregation appears not to result from e.g.
  2. Obstruction of the vessel lumen can result from platelet adhesion (platelet buildup in damaged vessel lining) or platelet aggregation.

Blood Vessel

  1. With injury to a blood vessel, two processes occur: platelet aggregation and coagulation.

Binding

  1. Ristocetin induces platelet aggregation by inducing von Willebrand protein binding to the platelet GP Ib complex.

Type

  1. Platelet aggregation in this type of thrombophlebitis usually can be decreased with anti-inflammatory medications.

Hypercholesterolemia

  1. This result suggests that hypercholesterolemia causes decreased NO-dependent inhibition of platelet aggregation due to reduced NO utilization.

Metabolites

  1. These metabolites in turn activate the thromboxane receptor, and this predisposes these individuals towards platelet aggregation and thrombosis (clotting).

Fibrin

  1. Fibrin, in conjunction with the platelet aggregation, forms a mesh at the clot site.

Argatroban

  1. Argatroban had no influence on chromogenic protein C measurement, batroxobin time and induced platelet aggregation.

Cardiovascular Disease

  1. Platelet aggregation can contribute to cardiovascular disease which is the number one cause of death in the western world.

Pravastatin

  1. Policosanol more effectively reduced platelet aggregation than did Pravastatin, another important risk factor in cardiovascular disease.

Thrombus Formation

  1. Fifteen minutes after the injury, platelet aggregation and thrombus formation are observed.
  2. An antiplatelet drug is a member of a class of pharmaceuticals that decreases platelet aggregation and inhibits thrombus formation.

Thrombin

  1. Thrombin is the most potent platelet activator, which leads to platelet aggregation and the development of blood clots.
  2. Tyrosine phosphorylation of multiple platelet proteins is stimulated by thrombin and other agonists that cause platelet aggregation and secretion.
  3. For complete and reliable suppression of thrombus formation simultaneous administration of thrombin and platelet aggregation inhibitors is necessary.

Clot Formation

  1. The processes of clot formation and platelet aggregation terminate when stimuli for clot formation dissipate.
  2. Healthy endothelial function promotes vasodilation and inhibits platelet aggregation (clot formation).

Activation

  1. The delay between triggering and activation helps explain a decrease in platelet aggregation at higher flow rates.

Blood Coagulation

  1. They affect platelet aggregation, blood coagulation, fibrinolysis, complement system, blood pressure and nervous system.
  2. The process of platelet aggregation and blood coagulation are summarized in Figures 54–1 and 54–2.
  3. Fibrinogen strongly affects blood coagulation, blood rheology and platelet aggregation.

Treatment

  1. Treatment with aspirin has become common because of its positive effects on platelet aggregation.

Prostacyclin

  1. The document's authors said Vioxx might reduce the production of a substance called prostacyclin, which prevents platelet aggregation.

Etoricoxib

  1. Etoricoxib did not interfere with the antiplatelet effects of low-dose ASA, as assessed by serum TXB2 and platelet aggregation.

Lipid Profile

  1. Assessments of lipid profile, platelet aggregation tests, safety indicators and adverse events were performed.

Arachidonic

  1. Policosanol also decreases arachidonic acid and collagen-induced platelet aggregation.

Effects

  1. Effects of penicillin G on platelet aggregation, release and adherence to collagen.
  2. Effects of acute, moderate alcohol consumption on human platelet aggregation in platelet-rich plasma and whole blood.

Humans

  1. Barrie SA, Wright JV, Pizzorno JE. Effects of garlic on platelet aggregation, serum lipids and blood pressure in humans.
  2. Tocotrienols were shown to cause a decrease in thromboxane levels in humans by 20 - 26% and a decrease in platelet aggregation.

Categories

  1. Policosanol
  2. Fibrinogen
  3. Platelets
  4. Treatment > Diagnosis > Blood Tests > Bleeding Time
  5. Healthy Volunteers

Related Keywords

    * Adp * Aggregation * Aspirin * Bleeding * Bleeding Time * Blood * Coagulation * Coagulation Time * Collagen * Effect * Fibrinogen * Formation * Healthy Volunteers * Inhibition * Patients * Platelet * Platelets * Policosanol * Study * Thromboxane * Vitro * Von Willebrand Factor * Vwf
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  Originally created: April 04, 2011.
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